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Fig. 5 | BMC Cell Biology

Fig. 5

From: Microvesicles derived from hypoxia/reoxygenation-treated human umbilical vein endothelial cells promote apoptosis and oxidative stress in H9c2 cardiomyocytes

Fig. 5

H/R-EMVs up-regulated the expression of p38 MAPK and JNK1/2 in H9c2 cells. a Representative images of immunoblots of p38 MAPK activation in each group. b JNK1/2 activation in H9c2 cells (phospho-JNK1/2). * p < 0.05, ** p < 0.01 versus Control; # p < 0.05 versus H/R-EMVs 10 μg/mL. c ROS content in Control-EMVs and H/R-EMVs. *** p < 0.001 versus Control-EMVs

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