Erratum to: Aurora kinase-C-T191D is constitutively active mutant

  • Jabbar Khan1, 2,

    Affiliated with

    • Sanaullah Khan3Email author,

      Affiliated with

      • Sobia Attaullah4,

        Affiliated with

        • Ijaz Ali5 and

          Affiliated with

          • Shahid N Khan3

            Affiliated with

            BMC Cell Biology201213:17

            DOI: 10.1186/1471-2121-13-17

            Received: 28 May 2012

            Accepted: 26 June 2012

            Published: 26 June 2012

            The original article was published in BMC Cell Biology 2012 13:8

            Retraction

            The editors regretfully retract the article [1] by Jabbar Khan, Sanaullah Khan, Sobia Attaullah, Ijaz Ali, and Shahid Khan (BMC Cell Biology 2012, 13:8) due to significant overlap with previously published article “Overexpression of Active Aurora-C Kinase Results in Cell Transformation and Tumour Formation” by Jabbar Khan, Frédéric Ezan, Jean-Yves Crémet, Alain Fautre, David Gilot, Marine Lambert, Christelle Benaud, Marie-Bérengère Troadec, and Claude Prigent (PLoS ONE 6(10): e26512. doi:10.1371/journal.pone.0026512). We apologise to all affected parties for the inconvenience caused.

            Notes

            Authors’ Affiliations

            (1)
            Department of Biological Sciences, Gomal University Dera Ismail Khan
            (2)
            Institute of Genetics and Development, University of Rennes1
            (3)
            Department of Zoology, Kohat University of Science and Technology
            (4)
            Department of Zoology, Islamia College Peshawar (A Public Sector University), University Campus
            (5)
            Institute of Biotechnology and Genetic Engineering, Khyber Pakhtunkhwa University of agriculture Peshawar

            References

            1. Jabbar Khan, Sanaullah Khan, Sobia Attaullah, Ijaz Ali, Shahid NIAZ Khan: Aurora kinase-C-T191D is constitutively active mutant. BMC Cell Biology. 2012, 13: 8-10.1186/1471-2121-13-8.View Article

            Copyright

            © Khan et al.; licensee BioMed Central Ltd. 2012

            This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://​creativecommons.​org/​licenses/​by/​2.​0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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